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Automated Perimetry
Carbonic Anhydrase Inhibitors
Intra Ocular Lens Power Calculation and Optic Biometry...
Visual Field Defects in Glaucoma
Visual Field Defect and Retinal Nerve Fiber Layer Defect in a Case of Optic Nerve Head Drusen...
Current Minimal Invasive Angle Procedures Without Implants for the Treatment of Glaucoma...
Intra Ocular Lens Power Calculation and Optic Biometry...
Automated Perimetry
Carbonic Anhydrase Inhibitors
Visual Field Defect and Retinal Nerve Fiber Layer Defect in a Case of Optic Nerve Head Drusen...
PureSee Kesintisiz Yüksek Kalitede Görüş
TJ-CEO 2017 , Vol 12 , Num 2
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Cystic Macular Edema Secondary to Prostaglandin Analog
Sibel İNAN1, Ersan ÇETİNKAYA2
1Yrd. Doç. Dr., Afyon Kocatepe Üniversitesi, Göz Hastalıkları, Afyonkarahisar - TÜRKİYE
2Asist. Dr., Afyon Kocatepe Üniversitesi, Göz Hastalıkları, Afyonkarahisar - TÜRKİYE
Cystoid macular edema (CME) can develop due to a variety of causes. It is also known that some medications can cause CME. Antiglacomatous agents from the group of prostaglandin analogues are among of these medications. A-82 year-old man with primary open angle glaucoma followed in our glaucoma department developed vision loss 3 months after the addition of a prostaglandin analog agent to his current anti-glaucoma treatment. An epiretinal membrane (ERM) and CME was diagnosed after optical coherence tomographic investigation. Although established ERM could be accused for CME, prostaglandin analog was discontinued because of development of visual symptoms 3 months after starting the prostaglandin analog, and his vision started to improve with recovery of CME beginning 1 week after discontinuation. Our case was presented here, because CME might be treated in other ways in case of failure to correct evaluation of the clinical picture and its importance for this reason. Keywords : Cystoid macular edema, epiretinal membrane, prostaglandin analog
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